A
Anonymous
Guest
Eh, je l'ai pas tapé !Persillette":2n58wwb5 a dit:Annaïg, je pense qu'il faut un peu choucouter notre Vari, sinon ça va mal tourner...
-
Bienvenue sur Végéweb, vous souhaitez rejoindre le reste de nos membres ? N'hésitez pas à vous inscrire dès aujourd'hui !
A
Anonymous
Guest
Quoi ? J'ai rien comprisVari":1mosyfh9 a dit:
Persillette
Jeune bulbe
- Inscrit
- 21/7/09
- Messages
- 57
- Score de réaction
- 0
Moi non plus, peut être vari a-t-il un serpent chez lui.Annaïg":2rrzc0r4 a dit:Quoi ? J'ai rien comprisVari":2rrzc0r4 a dit:
nasha
Avale du tofu
il est paléoanthropologue, je crois.
Si vous aviez ces publications svp:
Curhan, G.C. et al. (1993). A Prospective Study of Dietary Calcium and Other Nutrients and the Risk of Symptomatic Kidney Stones. New England Journal of Medicine, 238. 833-8.
Stadler, K.M. (1997). The Diet and Cancer Connection. Virginia Cooperative Extension Publications, Virginia Tech. 348:141.
Sellmeyer, D.E., Stone, K.L., Sebastian, A., & Cummings, S.R. (2001). A high ratio of dietary animal to vegetable protein increases the rate of bone loss and the risk of fracture in postmenopausal women. Am J Clin Nutr. 73:118-22.
Merci!
Curhan, G.C. et al. (1993). A Prospective Study of Dietary Calcium and Other Nutrients and the Risk of Symptomatic Kidney Stones. New England Journal of Medicine, 238. 833-8.
Stadler, K.M. (1997). The Diet and Cancer Connection. Virginia Cooperative Extension Publications, Virginia Tech. 348:141.
Sellmeyer, D.E., Stone, K.L., Sebastian, A., & Cummings, S.R. (2001). A high ratio of dietary animal to vegetable protein increases the rate of bone loss and the risk of fracture in postmenopausal women. Am J Clin Nutr. 73:118-22.
Merci!
yuya
Mange de la salade
ouaouh! le sujet m'a l'air passionnant!!!
vari, bienvenu!
il est intéressant de pouvoir parler de tout ça, exposer ses points de vues avec des personnes venant d'un autre "milieu", ayant d'autres centres d'intérets...
si tout le monde a le même point de vue, on tourne en rond dans la discussion!!!
et il a des arguments qui se valent...
je ne participerais pas à la discussion présente, j'ai jamais su ordonner mes idées, ni argumenter...
vari, bienvenu!
il est intéressant de pouvoir parler de tout ça, exposer ses points de vues avec des personnes venant d'un autre "milieu", ayant d'autres centres d'intérets...
si tout le monde a le même point de vue, on tourne en rond dans la discussion!!!
et il a des arguments qui se valent...
je ne participerais pas à la discussion présente, j'ai jamais su ordonner mes idées, ni argumenter...
Si je suis là c'est parce qu'il y a une petite histoire pour ceux qui ont suivi.
Je repasserais de temps en temps voir les nouvelles du front
Si vous saviez comment je vais déguster mon neotheropode au menu se soir hum vous ratez quelques chose tout de meme
Je repasserais de temps en temps voir les nouvelles du front
Si vous saviez comment je vais déguster mon neotheropode au menu se soir hum vous ratez quelques chose tout de meme
yuya
Mange de la salade
ça c'est de la provoque !!! ^^
pendant que tu dégusteras ton neotheropode (faut que j'aille voir ce que c'est ^^) je ferais des papouilles à mes deux lapins de compagnie, tout en salivant à l'avance à l'idée de déguster de délicieux pierogi...
pendant que tu dégusteras ton neotheropode (faut que j'aille voir ce que c'est ^^) je ferais des papouilles à mes deux lapins de compagnie, tout en salivant à l'avance à l'idée de déguster de délicieux pierogi...
yuya
Mange de la salade
j'ai pas tout suivi, tu manges des dinos???
flolalapine
Massacre des légumes
AAAAAAAAH
Ca c'est mon domaine
Cancer Causes Control. 2009 Aug 14.
Meat intake, meat mutagens and risk of lung cancer in Uruguayan men.
De Stefani E, Boffetta P, Deneo-Pellegrini H, Ronco AL, Aune D, Acosta G, Brennan P, Mendilaharsu M, Ferro G.
Grupo de Epidemiología, Departamento de Anatomía Patológica, Facultad de Medicina, Hospital de Clínicas, Avenida Brasil 3080 department 402, 11300, Montevideo, Uruguay, [email protected].
OBJECTIVE: To determine the role of meat consumption and related mutagens in the etiology of lung cancer, we conducted a case-control study among Uruguayan males in the time period 1996-2004. METHODS: The study included 846 cases and 846 controls, frequency matched on age and residence. Both series were drawn from the four major public hospitals in Montevideo, Uruguay. Unconditional logistic regression was used to estimate odds ratios (ORs) and 95% confidence intervals (95% CIs) of lung cancer by quartiles of meat intake and mutagens. RESULTS: The highest vs. the lowest quartile of intake of total meat (OR = 2.04, 95% CI 1.42-2.92), red meat (OR = 2.33, 95% CI 1.63-3.32), and processed meat (OR = 1.79, 95% CI 1.22-2.65) was associated with increased risk of lung cancer, while intake of total white meat, poultry and fish was not. Heterocyclic amines (IQ, MeIQx, PhIP), nitrosamines and benzo[a]pyrene were directly associated with the risk of lung cancer (OR for PhIP 2.16, 95% CI 1.48-3.15). Moreover, both red meat and meat mutagens displayed higher risks among former smokers compared with current smokers. CONCLUSIONS: This study suggests that red and processed meat and meat mutagens may play a role in the etiology of lung cancer.
Ca c'est mon domaine
Cancer Causes Control. 2009 Aug 14.
Meat intake, meat mutagens and risk of lung cancer in Uruguayan men.
De Stefani E, Boffetta P, Deneo-Pellegrini H, Ronco AL, Aune D, Acosta G, Brennan P, Mendilaharsu M, Ferro G.
Grupo de Epidemiología, Departamento de Anatomía Patológica, Facultad de Medicina, Hospital de Clínicas, Avenida Brasil 3080 department 402, 11300, Montevideo, Uruguay, [email protected].
OBJECTIVE: To determine the role of meat consumption and related mutagens in the etiology of lung cancer, we conducted a case-control study among Uruguayan males in the time period 1996-2004. METHODS: The study included 846 cases and 846 controls, frequency matched on age and residence. Both series were drawn from the four major public hospitals in Montevideo, Uruguay. Unconditional logistic regression was used to estimate odds ratios (ORs) and 95% confidence intervals (95% CIs) of lung cancer by quartiles of meat intake and mutagens. RESULTS: The highest vs. the lowest quartile of intake of total meat (OR = 2.04, 95% CI 1.42-2.92), red meat (OR = 2.33, 95% CI 1.63-3.32), and processed meat (OR = 1.79, 95% CI 1.22-2.65) was associated with increased risk of lung cancer, while intake of total white meat, poultry and fish was not. Heterocyclic amines (IQ, MeIQx, PhIP), nitrosamines and benzo[a]pyrene were directly associated with the risk of lung cancer (OR for PhIP 2.16, 95% CI 1.48-3.15). Moreover, both red meat and meat mutagens displayed higher risks among former smokers compared with current smokers. CONCLUSIONS: This study suggests that red and processed meat and meat mutagens may play a role in the etiology of lung cancer.
flolalapine
Massacre des légumes
Et:
J Natl Cancer Inst. 2009 Jul 15;101(14):1001-11. Epub 2009 Jun 26.
Comment in:
J Natl Cancer Inst. 2009 Jul 15;101(14):972-3.
Dietary fatty acids and pancreatic cancer in the NIH-AARP diet and health study.
Thiébaut AC, Jiao L, Silverman DT, Cross AJ, Thompson FE, Subar AF, Hollenbeck AR, Schatzkin A, Stolzenberg-Solomon RZ.
Nutritional Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD, USA.
BACKGROUND: Previous research relating dietary fat, a modifiable risk factor, to pancreatic cancer has been inconclusive. METHODS: We prospectively analyzed the association between intakes of fat, fat subtypes, and fat food sources and exocrine pancreatic cancer in the National Institutes of Health-AARP Diet and Health Study, a US cohort of 308 736 men and 216 737 women who completed a 124-item food frequency questionnaire in 1995-1996. Hazard ratios (HRs) and 95% confidence intervals (CIs) were calculated using Cox proportional hazards regression models, with adjustment for energy intake, smoking history, body mass index, and diabetes. Statistical tests were two-sided. RESULTS: Over an average follow-up of 6.3 years, 865 men and 472 women were diagnosed with exocrine pancreatic cancer (45.0 and 34.5 cases per 100 000 person-years, respectively). After multivariable adjustment and combination of data for men and women, pancreatic cancer risk was directly related to the intakes of total fat (highest vs lowest quintile, 46.8 vs 33.2 cases per 100 000 person-years, HR = 1.23, 95% CI = 1.03 to 1.46; P(trend) = .03), saturated fat (51.5 vs 33.1 cases per 100 000 person-years, HR = 1.36, 95% CI = 1.14 to 1.62; P(trend) < .001), and monounsaturated fat (46.2 vs 32.9 cases per 100 000 person-years, HR = 1.22, 95% CI = 1.02 to 1.46; P(trend) = .05) but not polyunsaturated fat. The associations were strongest for saturated fat from animal food sources (52.0 vs 32.2 cases per 100 000 person-years, HR = 1.43, 95% CI = 1.20 to 1.70; P(trend) < .001); specifically, intakes from red meat and dairy products were both statistically significantly associated with increased pancreatic cancer risk (HR = 1.27 and 1.19, respectively). CONCLUSION: In this large prospective cohort with a wide range of intakes, dietary fat of animal origin was associated with increased pancreatic cancer risk.
J Natl Cancer Inst. 2009 Jul 15;101(14):1001-11. Epub 2009 Jun 26.
Comment in:
J Natl Cancer Inst. 2009 Jul 15;101(14):972-3.
Dietary fatty acids and pancreatic cancer in the NIH-AARP diet and health study.
Thiébaut AC, Jiao L, Silverman DT, Cross AJ, Thompson FE, Subar AF, Hollenbeck AR, Schatzkin A, Stolzenberg-Solomon RZ.
Nutritional Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD, USA.
BACKGROUND: Previous research relating dietary fat, a modifiable risk factor, to pancreatic cancer has been inconclusive. METHODS: We prospectively analyzed the association between intakes of fat, fat subtypes, and fat food sources and exocrine pancreatic cancer in the National Institutes of Health-AARP Diet and Health Study, a US cohort of 308 736 men and 216 737 women who completed a 124-item food frequency questionnaire in 1995-1996. Hazard ratios (HRs) and 95% confidence intervals (CIs) were calculated using Cox proportional hazards regression models, with adjustment for energy intake, smoking history, body mass index, and diabetes. Statistical tests were two-sided. RESULTS: Over an average follow-up of 6.3 years, 865 men and 472 women were diagnosed with exocrine pancreatic cancer (45.0 and 34.5 cases per 100 000 person-years, respectively). After multivariable adjustment and combination of data for men and women, pancreatic cancer risk was directly related to the intakes of total fat (highest vs lowest quintile, 46.8 vs 33.2 cases per 100 000 person-years, HR = 1.23, 95% CI = 1.03 to 1.46; P(trend) = .03), saturated fat (51.5 vs 33.1 cases per 100 000 person-years, HR = 1.36, 95% CI = 1.14 to 1.62; P(trend) < .001), and monounsaturated fat (46.2 vs 32.9 cases per 100 000 person-years, HR = 1.22, 95% CI = 1.02 to 1.46; P(trend) = .05) but not polyunsaturated fat. The associations were strongest for saturated fat from animal food sources (52.0 vs 32.2 cases per 100 000 person-years, HR = 1.43, 95% CI = 1.20 to 1.70; P(trend) < .001); specifically, intakes from red meat and dairy products were both statistically significantly associated with increased pancreatic cancer risk (HR = 1.27 and 1.19, respectively). CONCLUSION: In this large prospective cohort with a wide range of intakes, dietary fat of animal origin was associated with increased pancreatic cancer risk.
flolalapine
Massacre des légumes
Allez encore un:
Am J Gastroenterol. 2009 May;104(5):1231-40. Epub 2009 Apr 14.
Dietary meat intake in relation to colorectal adenoma in asymptomatic women.
Ferrucci LM, Sinha R, Graubard BI, Mayne ST, Ma X, Schatzkin A, Schoenfeld PS, Cash BD, Flood A, Cross AJ.
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland, USA. [email protected]
OBJECTIVES: No previous study has concurrently assessed the associations between meat intake, meat-cooking methods and doneness levels, meat mutagens (heterocyclic amines (HCAs) and polycyclic aromatic hydrocarbons), heme iron, and nitrite from meat and colorectal adenoma in asymptomatic women undergoing colonoscopy. METHODS: Of the 807 eligible women in a cross-sectional multicenter colonoscopy screening study, 158 prevalent colorectal adenoma cases and 649 controls satisfactorily completed the validated food frequency and meat questionnaires. Using an established meat mutagen database and new heme iron and nitrite databases, we comprehensively investigated the components of meat that may be involved in carcinogenesis. Using logistic regression, we estimated odds ratios (ORs) and 95% confidence intervals (CIs) within quartiles of meat-related variables. RESULTS: Red meat was associated positively with colorectal adenoma (OR fourth vs. first quartile = 2.02; 95% CI = 1.06-3.83; P trend = 0.38). Intake of pan-fried meat (OR = 1.72; 95% CI = 0.96-3.07; P trend = 0.01) and the HCA: 2-amino-3,8-dimethylimidazo[4,5-f]quinoxaline (MeIQx) (OR = 1.90; 95% CI = 1.05-3.42; P trend = 0.07) were also associated with an increased risk of colorectal adenoma. The new databases yielded lower estimates of heme iron and nitrite than previous assessment methods, although the two methods were highly correlated for both exposures. Although not statistically significant, there were positive associations between iron and heme iron from meat and colorectal adenoma. CONCLUSIONS: In asymptomatic women undergoing colonoscopy, colorectal adenomas were associated with high intake of red meat, pan-fried meat, and the HCA MeIQx. Other meat-related exposures require further investigation.
Am J Gastroenterol. 2009 May;104(5):1231-40. Epub 2009 Apr 14.
Dietary meat intake in relation to colorectal adenoma in asymptomatic women.
Ferrucci LM, Sinha R, Graubard BI, Mayne ST, Ma X, Schatzkin A, Schoenfeld PS, Cash BD, Flood A, Cross AJ.
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland, USA. [email protected]
OBJECTIVES: No previous study has concurrently assessed the associations between meat intake, meat-cooking methods and doneness levels, meat mutagens (heterocyclic amines (HCAs) and polycyclic aromatic hydrocarbons), heme iron, and nitrite from meat and colorectal adenoma in asymptomatic women undergoing colonoscopy. METHODS: Of the 807 eligible women in a cross-sectional multicenter colonoscopy screening study, 158 prevalent colorectal adenoma cases and 649 controls satisfactorily completed the validated food frequency and meat questionnaires. Using an established meat mutagen database and new heme iron and nitrite databases, we comprehensively investigated the components of meat that may be involved in carcinogenesis. Using logistic regression, we estimated odds ratios (ORs) and 95% confidence intervals (CIs) within quartiles of meat-related variables. RESULTS: Red meat was associated positively with colorectal adenoma (OR fourth vs. first quartile = 2.02; 95% CI = 1.06-3.83; P trend = 0.38). Intake of pan-fried meat (OR = 1.72; 95% CI = 0.96-3.07; P trend = 0.01) and the HCA: 2-amino-3,8-dimethylimidazo[4,5-f]quinoxaline (MeIQx) (OR = 1.90; 95% CI = 1.05-3.42; P trend = 0.07) were also associated with an increased risk of colorectal adenoma. The new databases yielded lower estimates of heme iron and nitrite than previous assessment methods, although the two methods were highly correlated for both exposures. Although not statistically significant, there were positive associations between iron and heme iron from meat and colorectal adenoma. CONCLUSIONS: In asymptomatic women undergoing colonoscopy, colorectal adenomas were associated with high intake of red meat, pan-fried meat, and the HCA MeIQx. Other meat-related exposures require further investigation.
flolalapine
Massacre des légumes
Bon honnêtement quand on fait des recherches sur le sujet "viande et cancer", il y a un consensus pour dire que la viande rouge en grande quantité est nocive. Par contre la consommation de poisson c'est protecteur, et la volaille c'est pas clair.
Pour le lait de vache il existe des études contradictoires, certains montrant un léger bénéfice sur la santé des personnes âgées (sur l'hypertension artérielle notamment) et d'autres suggèrant que c'est un facteur de risque pour le diabète à long terme.
Quand à l'ostéoporose, c'est beaucoup plus compliqué qu'une simple mesure de la masse osseuse à l'ostéodensitométrie. Les bonnes études qui se basent sur les fractures ne montrent pas de lien direct entre la consommation de calcium et le risque fracturaire (c'est plutôt la vitamine D qui compte)
Ex:
Food Nutr Res. 2008;52. doi: 10.3402/fnr.v52i0.1654. Epub 2008 Apr 2.
Calcium intake in elderly patients with hip fractures.
Cho K, Cederholm T, Lökk J.
Geriatric Department, Institution of Neurobiology, Caring Sciences and Society, Karolinska Institutet, Karolinska University Hospital, Huddinge, Sweden.
BACKGROUND: Dietary calcium intake is assumed important in the prevention and treatment of osteoporosis. However, people in countries with a high calcium intake from commodities such as milk and milk products have a high incidence of hip fracture. The effect and influence of calcium intake in the prevention of osteoporotic fracture vary from different studies. OBJECTIVE: To investigate premorbid daily calcium intake in patients with low energy hip fractures during four consecutive years. DESIGN: In total 120 patients (mean age 78+/-8.5 (SD) years) were included between 2002 and 2005. The patients answered a structured food frequency questionnaire (FFQ) and interviews on patients' daily calcium intake from food and supplements took place during a 6-month period before the fracture. Dual energy X-ray absorptiometry (DEXA) was performed in a subgroup of 15 patients. RESULTS: The mean daily calcium intake from food and supplementation was 970+/-500 mg. However, 38% of patients had an intake below the recommended 800 mg/day. There was no significant relationship between calcium intake and age, gender, bone mineral density, serum calcium or albumin, type of fracture or body mass index. The mean free plasma calcium concentration was 2.3+/-0.1, i.e. within the reference limit. In 2005, 80% of the patients who underwent DEXA had manifest osteoporosis. There was a trend towards decreased calcium intake over the observation period, with a mean calcium intake below 800 mg/day in 2005. CONCLUSIONS: Hip fracture patients had a mean calcium intake above the recommended daily intake, as assessed by a FFQ. However, more than one-third of patients had an intake below the recommended 800 mg/day. The intake appeared to decrease over the investigated years. The relationship between calcium intake and fracture susceptibility is complex.
Pour le lait de vache il existe des études contradictoires, certains montrant un léger bénéfice sur la santé des personnes âgées (sur l'hypertension artérielle notamment) et d'autres suggèrant que c'est un facteur de risque pour le diabète à long terme.
Quand à l'ostéoporose, c'est beaucoup plus compliqué qu'une simple mesure de la masse osseuse à l'ostéodensitométrie. Les bonnes études qui se basent sur les fractures ne montrent pas de lien direct entre la consommation de calcium et le risque fracturaire (c'est plutôt la vitamine D qui compte)
Ex:
Food Nutr Res. 2008;52. doi: 10.3402/fnr.v52i0.1654. Epub 2008 Apr 2.
Calcium intake in elderly patients with hip fractures.
Cho K, Cederholm T, Lökk J.
Geriatric Department, Institution of Neurobiology, Caring Sciences and Society, Karolinska Institutet, Karolinska University Hospital, Huddinge, Sweden.
BACKGROUND: Dietary calcium intake is assumed important in the prevention and treatment of osteoporosis. However, people in countries with a high calcium intake from commodities such as milk and milk products have a high incidence of hip fracture. The effect and influence of calcium intake in the prevention of osteoporotic fracture vary from different studies. OBJECTIVE: To investigate premorbid daily calcium intake in patients with low energy hip fractures during four consecutive years. DESIGN: In total 120 patients (mean age 78+/-8.5 (SD) years) were included between 2002 and 2005. The patients answered a structured food frequency questionnaire (FFQ) and interviews on patients' daily calcium intake from food and supplements took place during a 6-month period before the fracture. Dual energy X-ray absorptiometry (DEXA) was performed in a subgroup of 15 patients. RESULTS: The mean daily calcium intake from food and supplementation was 970+/-500 mg. However, 38% of patients had an intake below the recommended 800 mg/day. There was no significant relationship between calcium intake and age, gender, bone mineral density, serum calcium or albumin, type of fracture or body mass index. The mean free plasma calcium concentration was 2.3+/-0.1, i.e. within the reference limit. In 2005, 80% of the patients who underwent DEXA had manifest osteoporosis. There was a trend towards decreased calcium intake over the observation period, with a mean calcium intake below 800 mg/day in 2005. CONCLUSIONS: Hip fracture patients had a mean calcium intake above the recommended daily intake, as assessed by a FFQ. However, more than one-third of patients had an intake below the recommended 800 mg/day. The intake appeared to decrease over the investigated years. The relationship between calcium intake and fracture susceptibility is complex.
flolalapine
Massacre des légumes
Ah oui pour ceux qui se demandent de quoi je parle c'est de l'article:
A high ratio of dietary animal to vegetable protein increases the rate of bone loss and the risk of fracture in postmenopausal women
dans les materials and methods pour évaluer le risque fracturaire on se base sur la mesure de la densité osseuse en ostéodensitométrie, ce qui est un critère intermédiaire tout à fait merdique pour l'évaluation du risque fracturaire.
Et croyez-moi, je m'y connais en lecture critique.
A high ratio of dietary animal to vegetable protein increases the rate of bone loss and the risk of fracture in postmenopausal women
dans les materials and methods pour évaluer le risque fracturaire on se base sur la mesure de la densité osseuse en ostéodensitométrie, ce qui est un critère intermédiaire tout à fait merdique pour l'évaluation du risque fracturaire.
Et croyez-moi, je m'y connais en lecture critique.
flolalapine
Massacre des légumes
Attends je vais pas tout traduire tout de même...barbux":2e6pyk6u a dit:Flo, si tu comprends ce qui est écrit, peut-être pourrais-tu l'écrire en français ?
Feignasse!!!
flolalapine
Massacre des légumes
J'ai fait un résumé en +!!!flolalapine":ekwl5mqe a dit:Attends je vais pas tout traduire tout de même...barbux":ekwl5mqe a dit:Flo, si tu comprends ce qui est écrit, peut-être pourrais-tu l'écrire en français ?
Feignasse!!!
"Bon honnêtement quand on fait des recherches sur le sujet "viande et cancer", il y a un consensus pour dire que la viande rouge en grande quantité est nocive. Par contre la consommation de poisson c'est protecteur, et la volaille c'est pas clair.
Pour le lait de vache il existe des études contradictoires, certains montrant un léger bénéfice sur la santé des personnes âgées (sur l'hypertension artérielle notamment) et d'autres suggèrant que c'est un facteur de risque pour le diabète à long terme.
Quand à l'ostéoporose, c'est beaucoup plus compliqué qu'une simple mesure de la masse osseuse à l'ostéodensitométrie. Les bonnes études qui se basent sur les fractures ne montrent pas de lien direct entre la consommation de calcium et le risque fracturaire (c'est plutôt la vitamine D qui compte)"
Le premier abstract pourrais-je avoir l'article en entier svp? je doute de la méthodologie... Faire extrémement attention aux statistique qui peuvent révéler énormément de chose (voir la disparition du score de 400 au baseball) c'est pour quoi je souhaiterais lire l'article en entier.
De meme pour le deuxieme, excusez moi mais se baser sur des questionnaires remplis par l'etre humain me laisse perplexe, l'homme est un menteur née! "le risque de cancer du pancréas était directement liée à l'apport de gras total " et "865 hommes et 472 femmes" sur "308 736 hommes et 216 737 femmes" atteint d'un cancer du pancréas ca laisse encore plus perplexe non? et "Previous research relating dietary fat, a modifiable risk factor, to pancreatic cancer has been inconclusive."
Pareil pour le troisieme, "Although not statistically significant, there were positive associations between iron and heme iron from meat and colorectal adenoma. "
Quand au dernier il semble en effet que se ne soit pas clair ou plutot encore moins claire que pour les précédents...
L'homme peut mentir sur les questionnaires, donc ca fausse pas mal les résultats.
Le mieux serait de mettre 50 000 personnes dans des casiers avec caméra et tout le matériel de diagnostique, moitié végé moitié omni on verra bien ce que ca donne.
De meme pour le deuxieme, excusez moi mais se baser sur des questionnaires remplis par l'etre humain me laisse perplexe, l'homme est un menteur née! "le risque de cancer du pancréas était directement liée à l'apport de gras total " et "865 hommes et 472 femmes" sur "308 736 hommes et 216 737 femmes" atteint d'un cancer du pancréas ca laisse encore plus perplexe non? et "Previous research relating dietary fat, a modifiable risk factor, to pancreatic cancer has been inconclusive."
Pareil pour le troisieme, "Although not statistically significant, there were positive associations between iron and heme iron from meat and colorectal adenoma. "
Quand au dernier il semble en effet que se ne soit pas clair ou plutot encore moins claire que pour les précédents...
L'homme peut mentir sur les questionnaires, donc ca fausse pas mal les résultats.
Le mieux serait de mettre 50 000 personnes dans des casiers avec caméra et tout le matériel de diagnostique, moitié végé moitié omni on verra bien ce que ca donne.
flolalapine
Massacre des légumes
C'est le gros problème des études de nutrition, c'est bourré de biais.
Difficile d'imposer 30 ans de régime alimentaire à un être humain de manière randomisée et appariée.
Ceci dit je n'ai mis que quelques exemples. Fais une recherche sur Pubmed avec les mots clés "Meat intake" et "Cancer", tu verras le nombre d'études. Sujet à la mode.
Je précise en passant que je ne suis pas végé.
Difficile d'imposer 30 ans de régime alimentaire à un être humain de manière randomisée et appariée.
Ceci dit je n'ai mis que quelques exemples. Fais une recherche sur Pubmed avec les mots clés "Meat intake" et "Cancer", tu verras le nombre d'études. Sujet à la mode.
Je précise en passant que je ne suis pas végé.